The same problem can affect people whose sleep is frequently interrupted.Īs a result, chronic sleep deprivation has been linked to numerous heart problems including high blood pressure, high cholesterol, heart attack, obesity, diabetes, and stroke. Without sufficient nightly sleep, a person doesn’t spend enough time in the deep stages of NREM sleep that benefit the heart. These changes reduce stress on the heart, allowing it to recover from strain that occurs during waking hours. During the non-rapid eye movement (NREM) sleep stages, heart rate slows, blood pressure drops, and breathing stabilizes. Sleep is an essential time for the body to recuperate. Substantial evidence demonstrates that sleeping problems, including sleep deprivation and fragmented sleep, have negative effects on heart health. Does Sleep Deprivation Affect Heart Health? For the cardiovascular system, insufficient or fragmented sleep can contribute to problems with blood pressure and heighten the risk of heart disease, heart attacks, diabetes, and stroke.Īs a result, getting good sleep may help prevent damage to the cardiovascular system, and for people with heart problems, can be part of following a heart-healthy lifestyle. Sleep provides time for the body to restore and recharge, playing a key role in nearly all aspects of physical health. While it’s already well-known that factors like poor diet, limited exercise, and smoking can harm the heart, there’s a growing recognition of the dangers of sleep deprivation for heart health. Unfortunately, heart problems are a leading cause of illness and death in the United States. Responsible for pumping blood throughout the body, the heart powers the circulatory system that ensures that all the organs and tissues in the body get the oxygen they need. There is positive correlation between Troponin-I and BNP levels in first attack of AMI patients with acute heart failure.It’s difficult to overstate the heart’s importance to health. Thus, the present study shows that the higher the Troponin-I levels, the higher the BNP levels in first attack of AMI patients and the more severe the heart failure (more severe left ventricle dysfunction).
Echocardiography shows that patients with high BNP level has low ejection fraction (LVEF) and patients with low BNP level has preserved ejection fraction (LVEF). In this study, it was shown that the levels of BNP had positive correlation with Troponin-I levels, with medium strength of association (r=0.734, p<0.05). Mean Troponin-I of Group I and Group II were 3.10☒.68 and 62.93☓2.75ng/ml respectively & mean BNP value of Group I and Group II were 20.96☑4.18 and 615.65☒49.27pg/ml respectively.
Group I: Patients with first attack of acute myocardial infarction (without heart failure) & Group II: Patients with first attack of acute myocardial infarction with acute heart failure. Total 100 patients were studied and divided into two groups - 50 patients in each group. This cross-sectional analytical study was conducted in the Department of Cardiology in Mymensingh Medical College Hospital from January 2014 to December 2014. Rationale of this study is to see, whether quantitative Troponin alone can serve for both diagnosis and prognosis of AMI Patients with heart failure or not. In this study, we investigate the correlation of Troponin-I with BNP levels in patients presenting with AMI with or without Acute Heart Failure. But B-type natriuretic peptide (BNP) level is also elevated in AMI and is a quantitative biochemical marker related to the extent of infarction and the left ventricle systolic dysfunction. Troponins are regarded as markers of choice for the diagnosis of acute myocardial infarction (AMI).
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